Current Style: Standard
Shin YJ , Kim JH , Seo JM Protective effect of clusterin on oxidative stress-induced cell death of human corneal endothelial cells. Mol Vis. 2009 Dec 16;15:2789-95.
PURPOSE: To investigate the protective effect of clusterin on oxidative stress-induced cell death of human corneal endothelial cells.
THE ROLE OF MACROPHAGES IN OPTIC NERVE REGENERATION
Neuroscience. 2009 February 6; 158(3): 1039–1048.
Paresh posted an excellent message about dealing with glaucoma in the FitEyes.com forums. He asked seven great questions. I decided to do my best to respond to each question. I'm going to take them one at a time. Paresh's first question was , "what we can do in daily life to limit further damage to eyes?" This blog post is my first response.
This article is my attempt to unify some of what has been proposed about the mechanism underlying retinal ganglion cell (RGC) loss in glaucoma.
Glaucoma is an optic neuropathy in which retinal ganglion cells die probably through an apoptotic process. Apoptosis is known to involve free radicals in several systems including the retina. In this context, the aim of the present work was to analyze retinal oxidative damage in rats with glaucoma induced by the chronic injection of hyaluronic acid in the eye anterior chamber. The results showed a significant decrease in total retinal superoxide dismutase and catalase activities after 6 and 3 weeks of treatment with hyaluronic acid, respectively.
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